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Tara Seshadri

from Sturbridge, MA
Age ~63
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Also known as:
Tara Srinivasachar, Tara S Eshadri, Tara I, Vidhya Lakshminarasimh
Phone and address:
10 Podunk Rd, Fiskdale, MA 01566
(508) 347-2933
Related to:
Nadathur S Seshadri
Connected to:
Ananth Seshadri, 35Jayshree Seshadri, 49Sahil Seshadri, 39Sudhir Seshagiri, 53Vidya Seshan, 50Godwin O Seshie, 73Nicole K Sesi, 38Vanessa Sesi, 42John L Sesini, 62Adam K Sesker, 37

Tara Seshadri Phones & Addresses

  • 10 Podunk Rd, Sturbridge, MA 01566 (508) 347-2933 (508) 347-9335
  • Littleton, MA
  • Malden, MA
  • Albany, NY
  • 10 Podunk Rd, Sturbridge, MA 01566 (508) 328-0528

Work

Company: AbbVie Bioresearch Center Inc. Address: 100 Research Dr, Worcester, MA 01605 Specialities: Intellectual Property - 100%

Education

Degree: Bachelor's degree or higher

Ranks

Licence: Massachusetts - Active Date: 2005

Emails

t***[email protected]

Professional Records

Lawyers & Attorneys

Tara Seshadri Photo 1

Tara Seshadri, Worcester MA - Lawyer

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Address:
AbbVie Bioresearch Center Inc.
100 Research Dr, Worcester, MA 01605
(508) 688-8058 (Office)
Licenses:
Massachusetts - Active 2005
Specialties:
Intellectual Property - 100%
Tara Seshadri Photo 2

Tara Seshadri, Worcester MA - Lawyer

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Address:
100 Research Dr, Worcester, MA 01605
Phone:
(508) 688-8058 (Phone)
Experience:
20 years
Jurisdiction:
Massachusetts (2005)
Memberships:
Massachusetts State Bar (2005)

Publications

Us Patents

Transgenic Knockout Mouse Having Functionally Disrupted Interleukin-1.Beta. Converting Enzyme Gene

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US Patent:
61004456, Aug 8, 2000
Filed:
Oct 20, 1997
Appl. No.:
8/954536
Inventors:
Tara Seshadri - Sturbridge MA
Ping Li - Wellesley MA
Hamish Allen - Shrewsbury MA
Subhashis Banerjee - Shrewsbury MA
Michael Paskind - Cambridge MA
Assignee:
BASF Aktiengesellschaft
International Classification:
C12N 500
C12N 1500
C07H 2104
A01K 67027
US Classification:
800 18
Abstract:
A transgenic mouse having somatic and germ cells in which at least one allele of an endogenous interleukin-1. beta. converting enzyme (ICE) gene is functionally disrupted is provided. The mouse may be heterozygous or, more preferably, homozygous for the ICE gene disruption. In homozygous mice, secretion of mature interleukin-1. beta. and interleukin-1. alpha. is substantially reduced relative to non-mutant mice. The mice of the invention can be used as positive controls to evaluate the efficacy of ICE inhibitors and to identify disease conditions that can be treated with ICE inhibitors. A transgenic mouse having functionally disrupted endogenous ICE genes but which has been reconstituted with a human ICE gene is also provided. This mouse can be used to identify agents that inhibit human ICE in vivo. Nucleic acid constructs for functionally disrupting an endogenous ICE gene in a host cell, recombinant vectors including the nucleic acid construct, and host cells into which the nucleic acid construct has been introduced are also encompassed by the invention.
Control profile
Tara Seshadri from Sturbridge, MA, age ~63 Get Report